What we do is test and assess for a lack of blood flow. For our purposes the subject material will be limited to the musculoskeletal system. Please, look through the site and discover!
Every procedure we use is a test and each one is held for several seconds. This time allotment allows for a physiological change to take place; the change we want is a movement of fluid in the affected area.
The lack of blood flow we are interested in is in the microcirculation caused by too high a pressure in the involved tissues; blood travels from a higher pressure area to a lower one. If the tissue pressure approaches that of systole in the local capillaries, then circulation is compromised.
Every patient is different and therefore, no two tests are really the same. Testing results vary in three main ways: they may have no effect, they may result in a positive sign after only one session, or they may lead to other tests.
Procedures often elicit a response from the patient leading the therapist to perform another test, then another, and so on. In these cases the assessment can be made, but not on the initial visit. it may take weeks until a definitive response is found.
If the testing has a positive outcome, the finding will be: the disability is due to a lack of blood flow in the area of complaint.
The tissues most often affected are the nerves along with their associated connective tissue. The procedures could therefore be referred to neurological tests.
This pathophysiology is often seen in chronic conditions. Because it is the nerves that are involved, almost any part of the body can be affected. Even tissues that are not normally innervated, may become so after injury. The inflammatory stage of healing involves angiogenesis and neurogenesis. Common sites of involvement include tendons, ligaments, joint capsuls, discs (endplates), intervertebral foramen, fascia and muscle, bone, large nerves and blood vessels.
The same pathophysiology may also result in inflammatory conditions. Through homeostasis the body will try to get blood into the hypoxic area through several mechanisms and also shift the metabolism of the affected cells to be more anerobic. The resulting adaptive changes may be exaggerated and may end in inflammation.
The ‘Manuscripts’ post contains several papers I have written on the subject; the ‘References’ section has over one hundred abstracts describing aspects of non vascular edema; ‘Procedures’ has over two hours of techniques.
It is interesting that these procedures are very similar to those that have existed in various cultures for thousands of years.
Again, feel free to look over the site.